Filtros : "American Journal of Physiology - Renal Physiology" Limpar

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  • Source: American Journal of Physiology - Renal Physiology. Unidades: HU, EEFE

    Subjects: NEFROLOGIA, INSUFICIÊNCIA RENAL CRÔNICA, EXERCÍCIO FÍSICO, GLOMÉRULOS RENAIS, ALBUMINÚRIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SANTANA, Davi A et al. Acute exercise does not impair renal function in nondialysis chronic kidney disease patients regardless of disease stage. American Journal of Physiology - Renal Physiology, v. 313, n. 2, p. F547-F552, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00131.2017. Acesso em: 15 maio 2024.
    • APA

      Santana, D. A., Poortmans, J. R., Dórea, E. L., Machado, J. B. de A., Fernandes, A. L., Sá-Pinto, A. L., et al. (2017). Acute exercise does not impair renal function in nondialysis chronic kidney disease patients regardless of disease stage. American Journal of Physiology - Renal Physiology, 313( 2), F547-F552. doi:10.1152/ajprenal.00131.2017
    • NLM

      Santana DA, Poortmans JR, Dórea EL, Machado JB de A, Fernandes AL, Sá-Pinto AL, Gualano B, Roschel H. Acute exercise does not impair renal function in nondialysis chronic kidney disease patients regardless of disease stage [Internet]. American Journal of Physiology - Renal Physiology. 2017 ; 313( 2): F547-F552.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00131.2017
    • Vancouver

      Santana DA, Poortmans JR, Dórea EL, Machado JB de A, Fernandes AL, Sá-Pinto AL, Gualano B, Roschel H. Acute exercise does not impair renal function in nondialysis chronic kidney disease patients regardless of disease stage [Internet]. American Journal of Physiology - Renal Physiology. 2017 ; 313( 2): F547-F552.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00131.2017
  • Source: American Journal of Physiology - Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      LEITE-DELLOVA, D. C. A. et al. Genomic and nongenomic dose-dependent biphasic effect of aldosterone on Na+/H+ exchanger in proximal S3 segment: role of cytosolic calcium. American Journal of Physiology - Renal Physiology, v. 295, n. 51, p. F1342-52, 2008Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00048.2008. Acesso em: 15 maio 2024.
    • APA

      Leite-Dellova, D. C. A., Oliveira-Souza, M., Malnic, G., & Aires, M. de M. (2008). Genomic and nongenomic dose-dependent biphasic effect of aldosterone on Na+/H+ exchanger in proximal S3 segment: role of cytosolic calcium. American Journal of Physiology - Renal Physiology, 295( 51), F1342-52. doi:10.1152/ajprenal.00048.2008
    • NLM

      Leite-Dellova DCA, Oliveira-Souza M, Malnic G, Aires M de M. Genomic and nongenomic dose-dependent biphasic effect of aldosterone on Na+/H+ exchanger in proximal S3 segment: role of cytosolic calcium [Internet]. American Journal of Physiology - Renal Physiology. 2008 ; 295( 51): F1342-52.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00048.2008
    • Vancouver

      Leite-Dellova DCA, Oliveira-Souza M, Malnic G, Aires M de M. Genomic and nongenomic dose-dependent biphasic effect of aldosterone on Na+/H+ exchanger in proximal S3 segment: role of cytosolic calcium [Internet]. American Journal of Physiology - Renal Physiology. 2008 ; 295( 51): F1342-52.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00048.2008
  • Source: American Journal of Physiology - Renal Physiology. Unidade: FM

    Subjects: ÓXIDO NÍTRICO (APLICAÇÕES TERAPÊUTICAS), NEFROPATIAS (TERAPIA;FISIOPATOLOGIA), REGRESSÃO

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    • ABNT

      FUJIHARA, Clarice K. et al. Short-term nitric oxide inhibition induces progressive nephropathy after regression of initial renal injury. American Journal of Physiology - Renal Physiology, v. 290, n. 3, p. F632-F640, 2006Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00259.2005. Acesso em: 15 maio 2024.
    • APA

      Fujihara, C. K., Sena, C. R., Malheiros, D. M. A. C., Mattar, A. L., & Zatz, R. (2006). Short-term nitric oxide inhibition induces progressive nephropathy after regression of initial renal injury. American Journal of Physiology - Renal Physiology, 290( 3), F632-F640. doi:10.1152/ajprenal.00259.2005
    • NLM

      Fujihara CK, Sena CR, Malheiros DMAC, Mattar AL, Zatz R. Short-term nitric oxide inhibition induces progressive nephropathy after regression of initial renal injury [Internet]. American Journal of Physiology - Renal Physiology. 2006 ; 290( 3): F632-F640.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00259.2005
    • Vancouver

      Fujihara CK, Sena CR, Malheiros DMAC, Mattar AL, Zatz R. Short-term nitric oxide inhibition induces progressive nephropathy after regression of initial renal injury [Internet]. American Journal of Physiology - Renal Physiology. 2006 ; 290( 3): F632-F640.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00259.2005
  • Source: American Journal of Physiology - Renal Physiology. Unidade: ICB

    Subjects: HISTOLOGIA, FISIOLOGIA

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    • ABNT

      CARRARO-LACROIX, Luciene Regina et al. Increased NHE1 expression is associated with serum deprivation-induced differentiation in immortalized rat proximal tubule cells. American Journal of Physiology - Renal Physiology, v. 291, p. F129-F139, 2006Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00290.2005. Acesso em: 15 maio 2024.
    • APA

      Carraro-Lacroix, L. R., Ramirez, M. A., Zorn, T. M. T., Rebouças, N. A., & Malnic, G. (2006). Increased NHE1 expression is associated with serum deprivation-induced differentiation in immortalized rat proximal tubule cells. American Journal of Physiology - Renal Physiology, 291, F129-F139. doi:10.1152/ajprenal.00290.2005
    • NLM

      Carraro-Lacroix LR, Ramirez MA, Zorn TMT, Rebouças NA, Malnic G. Increased NHE1 expression is associated with serum deprivation-induced differentiation in immortalized rat proximal tubule cells [Internet]. American Journal of Physiology - Renal Physiology. 2006 ; 291 F129-F139.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00290.2005
    • Vancouver

      Carraro-Lacroix LR, Ramirez MA, Zorn TMT, Rebouças NA, Malnic G. Increased NHE1 expression is associated with serum deprivation-induced differentiation in immortalized rat proximal tubule cells [Internet]. American Journal of Physiology - Renal Physiology. 2006 ; 291 F129-F139.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00290.2005
  • Source: American Journal of Physiology - Renal Physiology. Unidade: FM

    Assunto: FARMACOLOGIA

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    • ABNT

      GONÇALVES, Anderson Ricardo Roman et al. Renal expression of COX-2, ANG II, and AT(1) receptor in remnant kidney: strong renoprotection by therapy with losartan and a nonsteroidal anti-inflammatory. American Journal of Physiology - Renal Physiology, v. 286, p. F945-F954, 2004Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00238.2003. Acesso em: 15 maio 2024.
    • APA

      Gonçalves, A. R. R., Fujihara, C. K., Mattar, A. L., Malheiros, D. M. A. C., Noronha, I. de L., De Nucci, G., & Zatz, R. (2004). Renal expression of COX-2, ANG II, and AT(1) receptor in remnant kidney: strong renoprotection by therapy with losartan and a nonsteroidal anti-inflammatory. American Journal of Physiology - Renal Physiology, 286, F945-F954. doi:10.1152/ajprenal.00238.2003
    • NLM

      Gonçalves ARR, Fujihara CK, Mattar AL, Malheiros DMAC, Noronha I de L, De Nucci G, Zatz R. Renal expression of COX-2, ANG II, and AT(1) receptor in remnant kidney: strong renoprotection by therapy with losartan and a nonsteroidal anti-inflammatory [Internet]. American Journal of Physiology - Renal Physiology. 2004 ; 286 F945-F954.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00238.2003
    • Vancouver

      Gonçalves ARR, Fujihara CK, Mattar AL, Malheiros DMAC, Noronha I de L, De Nucci G, Zatz R. Renal expression of COX-2, ANG II, and AT(1) receptor in remnant kidney: strong renoprotection by therapy with losartan and a nonsteroidal anti-inflammatory [Internet]. American Journal of Physiology - Renal Physiology. 2004 ; 286 F945-F954.[citado 2024 maio 15 ] Available from: https://doi.org/10.1152/ajprenal.00238.2003

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